Parma XXIII International Echo Meeting
June 18-19-20, 2015
Dear colleagues and friends ,
we are glad to send you the Final Program of our Meeting.
This year there will be a somewhat new format with 2 days of Scientific Program and one half day dedicated to a Satellite Session on “ Science , Development , Peace “, with themes that are relevant to each one of us.
The Faculty in its core will remain the same, with some new important additions.
The Social Program and the Program for Accompanying Persons will follow the previous lines.
We will do our best to maintain or enhance the spirit and the atmosphere of the preceding Meetings !
“ We are preparing to have a great time together ! “
J Cardiol. 2015 Jan 5. pii: S0914-5087(14)00345-1. doi: 10.1016/j.jjcc.2014.11.011. [Epub ahead of print]
Echocardiographic screening for congenital heart disease in 8819 children: A report from local community events for children’s healthcare.
Nishio S1, Kusunose K2, Yamada H3, Yamao M1, Hirata Y1, Mori K4, Matsuoka S5, Sata M3.
Williams-Beuren Syndrome: Computed Tomography Imaging Review
Karuna M. Das,Tarek S. Momenah,Sven G. Larsson,Shehla Jadoon,
Abdullah S. Aldosary,Edward Y. Lee
Williams-Beuren syndrome (WBS) affects young infants and children. The underlying etiopathogenesis of this rare disease is due to the mutation of the elastin gene that is responsible for the elasticity of the arterial wall. As a result of inadequate elastin production, the major systemic arteries become abnormally rigid and can be manifested by an impediment to the blood flow.
PROPHYLAXIS of BACTERIAL ENDOCARDITIS
PROPHYLAXIS of BACTERIAL ENDOCARDITIS
If you have congenital heart disease, print out this information and give it to your physician. You can also download a PDF version of the wallet card from AHA.
Antibiotic therapy must be practiced for prophylaxis of the bacterial endocarditis, every tyme the patient must be take surgical manipulations as of diagnostic or therapeutic type, as is indicated in the attached outline.
ABSTRACT: Five members in three generations of a family were affected by a congenital heart disease. Four of them had mild or severe coarctation of the aorta (CoA), either isolated or in association with other cardiac defects. Fetal echocardiography allowed prenatal diagnosis in one pregnancy at risk. This family suggests that a rare form of CoA could be the result of an autosomal dominant mutation with high penetrance and variable expressivity rather than polygenic inheritance.
Journal of Medical Genetics 05/1993; 30(4):328-9. · 5.70 Impact Factor
ABSTRACT: Cardiac and extracardiac anomalies tend to follow characteristic patterns when there is an isomeric arrangement of the heart and lungs. We present a case diagnosed correctly during prenatal life as having isomerism of the left atrial appendages in which some of the associated features were most unexpected, namely absence of the spleen and obstructed totally anomalous pulmonary venous connection. This association proved misleading for us in the management of the patient. We found it difficult to believe our findings in the light of our past knowledge, and this resulted in a delay in the referral for surgery. The final outcome was unfortunately fatal. In the management of isomerism of any type, therefore, a wide spectrum of combinations and “crossover” of cardiac anomalies must be anticipated, emphasizing the need to assess individually the arrangement of the different organs of the body.
Cardiology in the Young 03/1994; 4(02):160 – 163. · 0.95 Impact Factor
Persistently low cardiac output predicts high mortality in newborns with cardiogenic shock.
Neonatal Intensive Care Unit, University of Chieti, Italy.
Panminerva medica (Impact Factor: 0.98). 03/1998; 40(1):28-32.
ABSTRACT Cardiogenic shock is an acute “unstable” state of circulatory dysfunction.
To evaluate the haemodynamic aspects of 45 newborn infants affected by cardiogenic shock, in relation to etiologies and the mortality rate.
Retrospective, correlation study of etiologies, mortality rate and haemodynamic measurements by Doppler ultrasound parameters.
45 newborn infants with cardiogenic shock, Gestational Age (GA) 37 (34-41), Birth Weight (BW) 2750 (1600-3600) g and and a control group of 20 neonates [GA 38 (34-40) wks, BW 2960 (1750-3800) g].
Left ventricular Cardiac Output (CO) was measured by a duplex scanner with pulsed Doppler and color flow mapping echocardiography in the first 48 hours of life.
In healthy newborns CO was 240 mL/min/kg (210-280), in newborns with cardiogenic shock CO was 170 mL/min/kg (f130-200), p < 0.01. The Stroke Volume was 1.80 mL/kg (1.70-1.90) in the control group and 1.20 (1.15-1.80) in the shocked group (p < 0.05); Heart Rate was 146 beats/min (130-160) and 160 beats/mm (140-194), (p < 0.01) respectively. Twenty-nine infants with cardiogenic shock had CO < or = 150 mL/min/kg and 5 of them (17.2%) died; 16 neonates had CO > 150 mL/min/kg and only one (6.2%) died (p < .01). Twenty-six of 36 newborns with perinatal asphyxia had CO < or = mL/min/kg and 5 of them died, while only one of 10 infants with CO > 150 mL/min/kg died.
Our results show that persistently low CO (< or = mL/min/kg) during the first 48 hours life, in newborns with cardiogenic shock due to perinatal asphyxia is associated with a significantly higher mortality.